Search results for "brain injuries"

showing 10 items of 139 documents

Systemic PaO2 oscillations cause mild brain injury in a pig model

2016

OBJECTIVE: Systemic PaO2 oscillations occur during cyclic recruitment and derecruitment of atelectasis in acute respiratory failure and might harm brain tissue integrity. DESIGN: Controlled animal study. SETTING: University research laboratory. SUBJECTS: Adult anesthetized pigs. INTERVENTIONS: Pigs were randomized to a control group (anesthesia and extracorporeal circulation for 20 hr with constant PaO2, n = 10) or an oscillation group (anesthesia and extracorporeal circulation for 20 hr with artificial PaO2 oscillations [3 cycles min⁻¹], n = 10). Five additional animals served as native group (n = 5). MEASUREMENTS AND MAIN RESULTS: Outcome following exposure to artificial PaO2 oscillations…

Pulmonary Atelectasismedicine.medical_specialtySwinemedicine.medical_treatment10208 Institute of NeuropathologyHippocampusInflammation610 Medicine & healthHippocampal formationReal-Time Polymerase Chain ReactionCritical Care and Intensive Care Medicinegamma-Aminobutyric acidRNA ComplementaryRandom Allocation03 medical and health sciencesExtracorporeal Membrane Oxygenation0302 clinical medicine030202 anesthesiologyNeurotransmitter receptorInternal medicinemedicineExtracorporeal membrane oxygenationAnimalsReceptoralpha-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic Acidgamma-Aminobutyric AcidRespiratory Distress Syndromebusiness.industryExtracorporeal circulationRespiration Artificialrespiratory tract diseasesEndocrinologyBrain Injuries570 Life sciences; biologyBlood Gas AnalysisInflammation Mediatorsmedicine.symptombusiness2706 Critical Care and Intensive Care Medicine030217 neurology & neurosurgerymedicine.drug
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Discovery of an epidermoid of the skull concomitant with a homolateral subacute traumatic subdural hematoma

1979

The case of a man of 32 years with an epidermoid of the left side of the skull is reported. The tumor itself was asymptomatic and was discovered accidentally because of a subdural hematoma on the same side. After a closed head injury, this patient had symptoms of an intracranial space occupying lesion (hematoma) on the left after a symptom-free interval. The left carotid angiogram demonstrated the characteristic picture of a subdural hematoma in the left parieto-temporo-occipital region. At the same time, characteristic changes in the skull on the same side, which were more apparent after removal of the hematoma, suggested an epidermal tumour of the skull. This was verified at operation.

AdultMalemedicine.medical_specialtyNeurologyAsymptomaticHematomaotorhinolaryngologic diseasesmedicineHumanscardiovascular diseasesCholesteatomaNeuroradiologybusiness.industrySkullHead injurymedicine.diseaseSurgerybody regionsSkullHematoma Subduralsurgical procedures operativemedicine.anatomical_structureNeurologyBrain InjuriesConcomitantClosed head injurycardiovascular systemNeurology (clinical)medicine.symptombusinessJournal of Neurology
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Skull Fractures Induce Neuroinflammation and Worsen Outcomes after Closed Head Injury in Mice

2020

The weight-drop model is used widely to replicate closed-head injuries in mice; however, the histopathological and functional outcomes may vary significantly between laboratories. Because skull fractures are reported to occur in this model, we aimed to evaluate whether these breaks may influence the variability of the weight-drop (WD) model. Male Swiss Webster mice underwent WD injury with either a 2 or 5 mm cone tip, and behavior was assessed at 2 h and 24 h thereafter using the neurological severity score. The expression of interleukin (IL)-6, IL-1β, tumor necrosis factor-α, matrix metalloproteinase-9, and tissue inhibitor of metalloproteinase-1 genes was m…

Male030506 rehabilitationmedicine.medical_specialtyTraumatic brain injurySkull fractureMice03 medical and health sciencesTraumatic brain injury0302 clinical medicineNeuroinflammationSkull fractureHead Injuries ClosedBrain Injuries TraumaticWeight-drop modelmedicineAnimalsNeuroinflammationInflammationSkull Fracturesbusiness.industryOriginal Articlesmedicine.diseaseSurgeryDisease Models AnimalSkullmedicine.anatomical_structureClosed head injuryNeurology (clinical)0305 other medical sciencebusiness030217 neurology & neurosurgeryJournal of Neurotrauma
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Dietary salt promotes ischemic brain injury and is associated with parenchymal migrasome formation

2018

Sodium chloride promotes vascular fibrosis, arterial hypertension, pro-inflammatory immune cell polarization and endothelial dysfunction, all of which might influence outcomes following stroke. But despite enormous translational relevance, the functional importance of sodium chloride in the pathophysiology of acute ischemic stroke is still unclear. In the current study, we show that high-salt diet leads to significantly worse functional outcomes, increased infarct volumes, and a loss of astrocytes and cortical neurons in acute ischemic stroke. While analyzing the underlying pathologic processes, we identified the migrasome as a novel, sodium chloride-driven pathomechanism in acute ischemic …

0301 basic medicineMalePathologyMacroglial CellsSodium ChlorideVascular MedicineBrain IschemiaMice0302 clinical medicineCytosolAnimal CellsMedicine and Health SciencesMedicineEndothelial dysfunctionStrokeNeuronsCerebral CortexCerebral IschemiaMultidisciplinaryQRPathophysiologyStrokeChemistryNeurologyPhysical SciencesImmunohistochemistryMedicineCellular Structures and OrganellesCellular TypesIntracellularResearch Articlemedicine.medical_specialtyScienceCerebrovascular DiseasesGlial Cells03 medical and health sciencesImmune systemIn vivoParenchymaAnimalscardiovascular diseasesVesiclesSodium Chloride DietaryMicroglial CellsNutritionIschemic StrokeOrganellesbusiness.industryChemical CompoundsBiology and Life SciencesCell Biologymedicine.diseaseDiet030104 developmental biologyCellular NeuroscienceAstrocytesBrain InjuriesSaltsbusiness030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Executive function and metacognitive self-awareness after severe traumatic brain injury.

2008

The objective of this study is to identify the clinical, neuropsychological, neuropsychiatric, and functional variables that correlate with metacognitive self-awareness (SA) in severe traumatic brain injury (TBI) outpatients and to assess the influence of the same variables on the sensory-motor, cognitive, and behavioral-affective indicators of SA. This cross-sectional observational study evaluated 37 outpatients from May 2006 to June 2007 in a neurorehabilitation hospital on the basis of the following inclusion criteria: (1) age ≥ 15 years; (2) diagnosis of severe TBI (Glasgow Coma Scale, GCS ≤ 8); (3) posttraumatic amnesia (PTA) resolution; (4) capacity to undergo formal psychometric eval…

AdultMalemedicine.medical_specialtyAdolescentTraumatic brain injurymedicine.medical_treatmentStatistics as TopicExecutive functions Metacognitive self-awareness Set shifting ability Perseverative responses TBI rehabilitationMetacognitionNeuropsychological TestsDisability EvaluationYoung AdultCognitionTBI rehabilitationExecutive functionAphasiaSurveys and QuestionnairesmedicineHumansPsychiatryNeurorehabilitationProblem SolvingRetrospective StudiesMetacognitive self-awareneRehabilitationSettore M-PSI/02 - Psicobiologia E Psicologia FisiologicaGeneral NeuroscienceSet shifting abilityNeuropsychologyPerseverative responseCognitionAwarenessExecutive functionsmedicine.diseaseSelf ConceptPsychiatry and Mental healthClinical PsychologyBrain InjuriesFemaleNeurology (clinical)medicine.symptomPsychology
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Xenon improves long-term cognitive function, reduces neuronal loss and chronic neuroinflammation, and improves survival after traumatic brain injury …

2019

Background.Xenon is a noble gas with neuroprotective properties. We previously showed that xenon improves short and long-term outcomes in young adult mice after controlled cortical impact (CCI). This is a follow-up study investigating xenon’s effect on very long-term outcome and survival. Methods.C57BL/6N (n=72) young adult male mice received single CCI or sham surgery and were treated with either xenon (75%Xe:25%O2) or control gas (75% N2:25%O2). The outcomes used were: 1) 24-hour lesion volume and neurological outcome score; 2)contextual fear-conditioning at 2 weeks and 20 months; 3) corpus callosum white matter quantification; 4) immunohistological assessment of neuroinflammation and neu…

MaleXenonhippocampusnerve degenerationCorpus callosumBUPRENORPHINEneuroinflammationMice0302 clinical medicineCognition030202 anesthesiologyAnesthesiologyBrain Injuries TraumaticMedicineEPIDEMIOLOGYYoung adultmemory disordersNeuronstraumatic brain injurySham surgeryBrain3. Good healthD-ASPARTATE RECEPTORmedicine.anatomical_structureNeuroprotective AgentsAnesthesianeuroprotectionmedicine.symptomLife Sciences & BiomedicineTraumatic brain injuryHYPOPITUITARISMNeuroprotectionWhite matter03 medical and health sciencesANALGESIAINHALED XENONAnimalsgeneral anaesthesiaSurvival analysisHYPOTHERMIAInflammationScience & Technologybusiness.industry1103 Clinical SciencesHypothermiamedicine.diseaseCOMPETITIVE-INHIBITIONSurvival AnalysisMice Inbred C57BLPATHOLOGYDisease Models AnimalAnesthesiology and Pain MedicineChronic DiseasebusinessCognition Disorders030217 neurology & neurosurgeryWHITE-MATTER DAMAGEFollow-Up StudiesBritish journal of anaesthesia
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Role of Sortilin in Models of Autoimmune Neuroinflammation

2015

Abstract The proneurotrophin receptor sortilin is a protein with dual functions, being involved in intracellular protein transport, as well as cellular signal transduction. The relevance of the receptor for various neuronal disorders, such as dementia, seizures, and brain injury, is well established. In contrast, little is known about the role of sortilin in immune cells and inflammatory diseases. The aim of our study was to elucidate the distribution of sortilin in different immune cell types in mice and humans and to analyze its function in autoimmune CNS inflammation. Sortilin was expressed most profoundly in murine and human macrophages and dendritic cells and to a much lesser extent in…

Central Nervous SystemCell typeEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisT-LymphocytesEncephalomyelitisImmunologyAutoimmunityBiologyMiceImmune systemmedicineAnimalsHumansImmunology and AllergyReceptorNeuroinflammationMice KnockoutAutoimmune diseaseAntigen PresentationMacrophagesExperimental autoimmune encephalomyelitisDendritic Cellsmedicine.diseaseImmunity InnateMice Inbred C57BLAdaptor Proteins Vesicular TransportBrain InjuriesImmunologyNeurogenic InflammationSignal transductionSignal Transduction
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Life goals after brain injury in the light of the dual process approach: empirical evidence and implications for neuropsychological rehabilitation.

2011

Sequelae of acquired brain injury endanger the realisation of important life-goals. Discrepancies arise between the importance attached to a goal and the success in realising it. This study investigates goal discrepancies and their influence on patients' subjective well-being (SWB) in different rehabilitation stages. Life-goals, SWB and daily functioning were assessed in 130 neurological inpatients and 42 outpatients by self-report questionnaires. Both patient groups reported greater discrepancies between importance and success of life-goals than a normative sample of healthy controls. In multiple regression modelling, goal discrepancy predicted SWB in the inpatient sample even when control…

AdultMalemedicine.medical_specialtymedicine.medical_treatmentPoison controlPersonal SatisfactionSeverity of Illness IndexOccupational safety and healthArts and Humanities (miscellaneous)Injury preventionActivities of Daily LivingmedicineHumansSubjective well-beingPsychiatryAcquired brain injuryApplied PsychologyPsychiatric Status Rating ScalesRehabilitationRehabilitationNeuropsychologyHuman factors and ergonomicsMiddle Agedmedicine.diseaseNeuropsychology and Physiological PsychologyBrain InjuriesCase-Control StudiesFemaleSelf ReportPsychologyGoalsClinical psychologyNeuropsychological rehabilitation
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Hyperosmolar therapy for acute brain injury: study protocol for an umbrella review of meta-analyses and an evidence mapping

2020

Introduction Acute brain injury is a challenging public health problem worldwide. Elevated intracranial pressure is a common complication after acute brain injury. Hyperosmolar therapy is one of the main therapeutic strategies for the management of intracranial hypertension. This study protocol outlines an umbrella review of meta-analyses which will investigate the benefits and harms of hyperosmolar therapy routinely used for the management of acute brain injury in the intensive care. Methods and analysis We will search PubMed/MEDLINE, EMBASE and the Cochrane Database of Systematic Reviews. We will include meta-analyses of primary research studies (eg, randomised controlled trials, observat…

meta-analysilaw.inventionAnaesthesia0302 clinical medicineHypertonic salinesystematic reviewlawBrain Injuries TraumaticProtocolMedicineMannitol1506030212 general & internal medicineBrain injuryRandomized Controlled Trials as TopicEvidence-Based MedicineRGeneral MedicineIntensive care unit3. Good healthTreatment OutcomeSystematic reviewResearch DesignMeta-analysisMedicinehyperosmolar therapy1682medicine.medical_specialtyCritical CareMEDLINE03 medical and health sciencesMeta-Analysis as TopicIntensive careHumansHyperosmolar therapyCerebral perfusion pressureIntensive care medicineSaline Solution HypertonicDroitbusiness.industrymannitolbrain injuryHypertonic salinemeta-analysisMeta-analysisSystematic reviewFluid TherapyObservational studyIntracranial Hypertensionbusiness030217 neurology & neurosurgeryhypertonic saline
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The role of mitochondrial transition pore, and its modulation, in traumatic brain injury and delayed neurodegeneration after TBI

2009

Following severe traumatic brain injury (TBI), a complex interplay of pathomechanism, such as exitotoxicity, oxidative stress, inflammatory events, and mitochondrial dysfunction occurs. This leads to a cascade of neuronal and axonal pathologies, which ultimately lead to axonal failure, neuronal energy metabolic failure, and neuronal death, which in turn determine patient outcome. For mild and moderate TBI, the pathomechanism is similar but much less frequent and ischemic cell death is unusual, except with mass lesions. Involvement of mitochondria in acute post-traumatic neurodegeneration has been extensively studied during the last decade, and there are a number of investigations implicatin…

Time FactorsTraumatic brain injurymedicine.medical_treatmentMitochondrionMitochondrial Membrane Transport ProteinsNeuroprotectionBrain Ischemiachemistry.chemical_compoundDevelopmental NeuroscienceCyclosporin aAnimalsHumansMedicineMitochondrial Permeability Transition Porebusiness.industryMPTPNeurodegenerationmedicine.diseasenervous system diseasesnervous systemNeurologyMitochondrial permeability transition porechemistryBrain InjuriesReperfusion InjuryAcute DiseaseChronic DiseaseNerve DegenerationAxotomybusinessNeuroscience
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